I can’t help but think all the tangentially positive stuff coming out about Ozempic is just marketing.
Ozempic has reduced the amount of manipulative marketing you see, too, and also your favorite dead childhood pet is somehow alive again and waiting at your back door thanks to Ozempic!
Is Ozempic what caused the Pet Sematery?
It is in the new A24 2027 reboot
Fyi the patent on GLP-1 has expired and there’s GLP-1 meds other than ozempic and wegovy rn
I feel the same but then again I did about statins and eye surgery.
I know this might be unrelated, so just a guess, but would people who are doing weekly injections, maybe bi-weekly or daily, also be more prone to take the flu shot and generally be medically more active, due to their inclination of trying to improve their weight/sugar levels using a medical injection. The article also mentions that this only proves correlation and not causation, which is hats off to gizmodo, but can be interesting if causation is proven in a future study
The big question here is whether GLP-1 is the cause or not being obese.
My neighbor believes it helps her ADHD and quit taking those meds.
That’s weird I feel more ADHD than ever.
I switched off vyvanse to Adderall because vyvanse was mostly for BED and wegovy is kicking BEDs ass. Adderall used to be much better for focus/concentration/prioritization/organization/etceteration. But now? I don’t think it’s working.
She had a really bad blood sugar episode and ended up in icu and she started ozempic a little after that. Said her sugar is more level than it’s ever been and her brain too. Think those are probably more related than she realized.
I thought I had anxiety for years but actually it was unmedicated ADHD and a bonus side effect is that my food noise is completely gone. I’ve lost 12lbs so far 😎
It would be great if the medical system had a way to see trends in data, if heading toward metabolic issues, chronic inflammation, before diabetes, types 2 and 3! Ahh, but fasting insulin and crp tests are cheap, don’t make much money for the system to prevent issues early, with lifestyle changes and not need treatments!
"Insulin resistance develops 10–20 years before fasting glucose or HbA1c cross into the prediabetic range. During that window, the pancreas compensates by pumping out progressively more insulin to keep glucose “normal”, which is what those markers capture.
HOMA-IR is the primary equation used to assess insulin resistance, calculated as (Fasting Insulin × Fasting Glucose) / 405, with values >2.0 indicate significant resistance and >2.9 metabolic syndrome. Triglyceride/HDL ratio is another marker for insulin resistance, with a target of <1.0 and high-risk values exceeding 3.0.
Fasting insulin levels are independently associated with high-sensitivity C-reactive protein (hs-CRP), a marker of systemic inflammation; higher fasting insulin predicts elevated CRP.
A nasty mechanism, the enzyme IDE (insulin-degrading enzyme) clears both insulin and Aβ. When hyperinsulinemia (from gut-driven peripheral IR) floods the system, IDE gets occupied by excess insulin and can’t clear Aβ as efficiently, plaques accumulate "*
- confirm for accuracy
